A low carb diet, specifically a low carb, high fat diet has incredible benefits. One of the benefits of going on the keto diet is its ability to fight against metabolic syndrome potentially. This syndrome affects the heart and can result in type 2 diabetes. However, there’s a solution, and it’s known as the keto diet. We discuss how this diet can help improve someone’s health in .the Body Reboot book. 

In Healthline, they provide a brief overview of metabolic syndrome. They also explain what symptoms to pay attention to if you’re experiencing this condition. 

Metabolic syndrome is a condition highly associated with your risk of diabetes and heart disease.

In fact, metabolic syndrome is a collection of symptoms, which include:

Abdominal obesity

Elevated blood pressure

Elevated fasting blood sugar levels

High triglycerides

Low “good” HDL cholesterol levels

However, a low-carb diet is incredibly effective in treating all five of these symptoms.

Under such a diet, these conditions are nearly eliminated.

In a study by Nutrition and Metabolism, researchers set out to find out if a low carb diet can prevent metabolic syndrome. They did find out that a low carb diet can be effective against preventing heart disease and other diseases. 

Metabolic Syndrome (MetS) represents a constellation of markers that indicates a predisposition to diabetes, cardiovascular disease and other pathologic states. The definition and treatment are a matter of current debate and there is not general agreement on a precise definition or, to some extent, whether the designation provides more information than the individual components. We consider here five indicators that are central to most definitions and we provide evidence from the literature that these are precisely the symptoms that respond to reduction in dietary carbohydrate (CHO). Carbohydrate restriction is one of several strategies for reducing body mass but even in the absence of weight loss or in comparison with low fat alternatives, CHO restriction is effective at ameliorating high fasting glucose and insulin, high plasma triglycerides (TAG), low HDL and high blood pressure. In addition, low fat, high CHO diets have long been known to raise TAG, lower HDL and, in the absence of weight loss, may worsen glycemic control. Thus, whereas there are numerous strategies for weight loss, a patient with high BMI and high TAG is likely to benefit most from a regimen that reduces CHO intake. Reviewing the literature, benefits of CHO restriction are seen in normal or overweight individuals, in normal patients who meet the criteria for MetS or in patients with frank diabetes. Moreover, in low fat studies that ameliorate LDL and total cholesterol, controls may do better on the symptoms of MetS. On this basis, we feel that MetS is a meaningful, useful phenomenon and may, in fact, be operationally defined as the set of markers that responds to CHO restriction. Insofar as this is an accurate characterization it is likely the result of the effect of dietary CHO on insulin metabolism. Glucose is the major insulin secretagogue and insulin resistance has been tied to the hyperinsulinemic state or the effect of such a state on lipid metabolism. The conclusion is probably not surprising but has not been explicitly stated before. The known effects of CHO-induced hypertriglyceridemia, the HDL-lowering effect of low fat, high CHO interventions and the obvious improvement in glucose and insulin from CHO restriction should have made this evident. In addition, recent studies suggest that a subset of MetS, the ratio of TAG/HDL, is a good marker for insulin resistance and risk of CVD, and this indicator is reliably reduced by CHO restriction and exacerbated by high CHO intake. Inability to make this connection in the past has probably been due to the fact that individual responses have been studied in isolation as well as to the emphasis of traditional therapeutic approaches on low fat rather than low CHO.

We emphasize that MetS is not a disease but a collection of markers. Individual physicians must decide whether high LDL, or other risk factors are more important than the features of MetS in any individual case but if MetS is to be considered it should be recognized that reducing CHO will bring improvement. Response of symptoms to CHO restriction might thus provide a new experimental criterion for MetS in the face of on-going controversy about a useful definition. As a guide to future research, the idea that control of insulin metabolism by CHO intake is, to a first approximation, the underlying mechanism in MetS is a testable hypothesis.

Similarly, another study, this time by NCBI: Metabolic Syndrome Related Disorder also discovered that this syndrome could be prevented by going on the keto diet. 

One of Robert Atkins contributions was to define a diet strategy in terms of an underlying metabolic principle (“the science behind Atkins”). The essential feature is that, by reducing insulin fluxes, lipids are funnelled away from storage and oxidized. Ketosis can be used as an indicator of lipolysis. A metabolic advantage is also proposed: controlled carbohydrates leads to greater weight loss per calorie than other diets. Although the Atkins diet and its scientific rationale are intended for a popular audience, the overall features are consistent with current metabolic ideas. We have used the Atkins controlled-carbohydrate diet as a focal point for teaching nutrition and metabolism in the first-year medical school curriculum. By presenting metabolism in the context of the current epidemic of obesity and of metabolic syndrome and related disorders, we provide direct application of the study of metabolic pathways, a subject not traditionally considered by medical students to be highly relevant to medical practice. We present here a summary of the metabolic basis of the Atkins diet as we teach it to medical students. We also discuss a proposed mechanism for metabolic advantage that is consistent with current ideas and that further brings out ideas in metabolism for students. The topics that are developed include the role of insulin and glucagon in lipolysis, control of lipoprotein lipase, the glucose-glycogen-gluconeogenesis interrelations, carbohydrate-protein interactions and ketosis. In essence, the approach is to expand the traditional feed-fast (post-absorptive) cycles to include the effect of low-carbohydrate meals: the disease states studied are generalized from traditional study of diabetes to include obesity and metabolic syndrome. The ideal diet for weight loss and treatment of metabolic syndrome, if it exists, remains to be determined, but presenting metabolism in the context of questions raised by the Atkins regimen prepares future physicians for critical analysis of clinical and basic metabolic information.

ABC News further explains why the keto diet works explicitly to control hunger and thus can prevent heart disease from forming in the future. 

A low-carbohydrate diet helps people with a condition called metabolic syndrome, a collection of serious risk factors found in some obese individuals.

Now, a new study confirms the diet is effective against the syndrome, and the researchers think they've discovered how it works.

Eating a low-carb diet improves the hormonal signaling involved in obesity and improves the sense of fullness, allowing weight loss, according to study leader Matthew R. Hayes, a postdoctoral fellow at the University of Pennsylvania.

“There is this strong interest in the field in carb-restricted diets in the treatment of obesity,” said Hayes, who conducted the research while a doctoral student at Pennsylvania State University. “That [interest] comes from a number of controlled clinical trials that demonstrate overweight or obese people, maintained on low-carb diets, are successful if they adhere to the diet.”

Hayes and his colleagues studied 20 men and women with metabolic syndrome, instructing them to follow a low-carb diet similar to the popular South Beach Diet. For phase one, which lasted two weeks, the study participants were told to get 10 percent of their calories from carbohydrates. For phase 2, which lasted the remaining 10 weeks of the study, they were told to eat up to 27 percent carbs.

“The subjects did lose weight, and they lost total body fat. Their weight was a little over 200 pounds when the study started. By the end of the study, the subjects weighed about 193, 194. They lost close to 10 pounds during the three-month study.”

And, Hayes said, “By the end of the study, about 50 percent no longer had metabolic syndrome.”

Researchers at The Ohio State University wanted to know what happens to obese people with metabolic syndrome, a precursor to diabetes, when they eat a diet low in carbohydrates but don't shed any pounds. They found that more than half of study participants no longer met the criteria for metabolic syndrome immediately following a four-week low-carb diet.

The new study included 16 men and women with metabolic syndrome, a cluster of factors that also put people at higher risk of heart disease and stroke. The conditions that contribute to metabolic syndrome include high blood pressure, high blood sugar, excess body fat around the waist and abnormally low ‘good' HDL cholesterol or high triglyceride levels. About a third of American adults have the syndrome, according to the American Heart Association.

After eating a low-carb diet, more than half the participants — five men and four women — saw their metabolic syndrome reversed even though they were fed diets that intentionally contained enough calories to keep their weight stable.

We wrote the Body Reboot book to encourage people to improve their health! It’s incredible to know that the keto diet has the potential to combat several diseases, including metabolic syndrome. Help us cover shipping and visit this page to get your free copy!

Sources: Healthline, Nutrition & Metabolism volume 2, Article number: 31 (2005), NCBI: Metab Syndr Relat Disord. 2003, ABC News, Science Daily/Ohio State University.